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These observations create a plausible mechanistic pathway in PCOS: metabolic and hormonal perturbations (hyperandrogenism, insulin resistance) promote platelet hyperreactivity and PMP release, which in turn primes neutrophils to NETose within adipose, ovarian, and vascular beds

doi: 10.1186/s12937-022-00812-x 69

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These simulations revealed the location where the receptor binding domain attaches to the ACE2 receptor a stretch of the ACE2 protein that forms a structure called an alpha helix

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